Aids and the Human Immune System
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Aids and the Human Immune System

What is it with the aids virus. Is it that it attacks the immune system or is it that it mutates so quickly that it wears down the immune system?

It is well known in warfare that one way to defeat a superior opponent is to use his own strategy to cause his self-defeat. 

So what are we dealing with here? An intellectual opponent who guesses our strategies? Well, it looks like a virus doesn't even have room for nuclear material let alone gray matter. It looks more like a knee jerk reaction of what it does best, and that's mutation.

By the way, this may be the precursor of a new order of microorganism. You may see airborne aids bacterium, water borne aids protozoans. Disguise the limit, as they say when they can't see it coming. There's been quite a bit of mutation going on these days. It's the name of the game, South African Penguins muscling in on human urban environments, Elk in playgrounds. You say that's behavioral? It's in the chemistry of the body that such reactions emerge and that's probable evidence of slight mutational changes. 

So the cure for aids may be in a different direction from all this viracide treatment and immune system boosting. Take a look at the aids survivors. You may see a reticence in immune system activity against the virus, thus drastically reducing the stimulus to mutate.

It may be therefore possible to reverse the immune strategy that is working against the macroorganism. We're talking controlled imunosupression, here. Use the body's identification and production systems but cause an interruption in the deployment of antibodies. Then once again, in lieu of looking to a robustness of an immune system, the emphasis is looking to the acceleration of the labeling and deployment aspect of the immune system to match and slightly surpass the mutation speed of the monocellular organism.  But if that is not yet possible, then it may be possible to withhold an attack against the virus that would only stimulate it's mutation until the key moment when the deployment would favor the host.

But you may find in research that the disease will not mutate without signals to do so. After all, that would seem to an organism to be counterproductive to survivability.

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